Limited Evidence On Best Next Seizure Medicine
This paper was a systematic review of randomized controlled trials about what to do after the first antiseizure medication (ASM) does not control focal epilepsy.
This hub covers pediatric epilepsy in infants, kids, and teens, including diagnosis, syndromes, development, school plans, and safety. New studies translated into clear takeaways for parents.
Usually when two appropriate medications haven’t controlled seizures.
Many families benefit and it depends on seizure frequency, medications, and learning needs.
Often yes, with smart precautions. Ask your neurologist or epileptologist about your child’s specific risks.
Clusters, prolonged seizures, breathing trouble, new weakness, or major regression.
This paper was a systematic review of randomized controlled trials about what to do after the first antiseizure medication (ASM) does not control focal epilepsy.
This study looked at adding ethosuximide, a seizure medicine, to treatment for children with developmental and epileptic encephalopathy with spike-wave activation in sleep (D/EE-SWAS).
This study followed 35 children and adolescents with epilepsy who started levetiracetam, a common seizure medicine.
This article discusses a proposed way to personalize dosing of perampanel, a seizure medicine used in drug-resistant epilepsy.
This study developed and tested a computer model called EffiFormer to detect epileptic seizures from EEG recordings.
Researchers looked at how long it took children with drug-resistant epilepsy to get a presurgical epilepsy evaluation after they were recognized as having drug-resistant epilepsy.
This report describes one infant with global developmental delay and refractory epilepsy associated with a TBC1D24 mutation.
This study looked at kidney stones and kidney calcium buildup in children with drug-resistant epilepsy who were treated with a ketogenic diet (KD) or a modified Atkins diet (MAD) at one hospital clinic.
This study looked at tubulinopathies, a group of neurodevelopmental disorders caused by changes in tubulin-related genes and microtubule-associated proteins.